🧪 Pharmacology Mastery

Know Your Drugs.

Mechanism of action, drug classification, and receptor pharmacology for every paramedic-level medication.

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💊 Quick Drug Reference

DrugClassificationMechanism of ActionKey Use
AdenosineAntiarrhythmicSlows AV nodal conduction via A1 receptor; transiently blocks AV nodeSVT conversion
AmiodaroneClass III AntiarrhythmicBlocks K+ channels → prolongs repolarization; also Na+, Ca2+, beta blockadeVF/VT, stable VT
AtropineAnticholinergicCompetitive muscarinic acetylcholine receptor antagonist → increases HRBradycardia, organophosphate OD
LidocaineClass Ib AntiarrhythmicBlocks fast Na+ channels → suppresses ventricular automaticityVF/VT (alternative)
Magnesium SulfateElectrolyte / AntiarrhythmicNMDA receptor antagonist; inhibits Ca2+ entry; membrane stabilizerTorsades, eclampsia, asthma
ProcainamideClass Ia AntiarrhythmicBlocks Na+ channels in both AV node and accessory pathwaysStable VT, WPW with A-Fib
EpinephrineCatecholamine / VasopressorAlpha-1 + Beta-1 + Beta-2 agonist → vasoconstriction, increased HR/contractility, bronchodilationCardiac arrest, anaphylaxis
NorepinephrineCatecholamine / VasopressorPredominantly Alpha-1 agonist → potent vasoconstriction; minimal Beta-2Septic/distributive shock
DopamineCatecholamine / VasopressorDose-dependent: dopaminergic (low) → Beta-1 (mid) → Alpha-1 (high)Cardiogenic shock, bradycardia
VasopressinAntidiuretic Hormone AnalogV1 receptor agonist → direct vascular smooth muscle constrictionCardiac arrest (alternative to epi)
SuccinylcholineDepolarizing NMBDepolarizes NMJ by mimicking ACh; persistent depolarization → paralysisRSI (intubation)
VecuroniumNon-Depolarizing NMBCompetitive ACh receptor antagonist at NMJ → non-depolarizing paralysisRSI maintenance paralysis
EtomidateImidazole / Sedative-HypnoticPotentiates GABA-A receptor → CNS depression; minimal cardiovascular effectRSI induction (hypotensive pts)
KetamineDissociative AnestheticNMDA receptor antagonist → dissociative state; catecholamine release → bronchodilationRSI (asthma), pain, agitation
MidazolamBenzodiazepinePotentiates GABA-A receptor → increased Cl- influx → CNS depressionSeizures, sedation, RSI pre-med
LorazepamBenzodiazepinePotentiates GABA-A receptor → increased Cl- influx → CNS depressionStatus epilepticus (IV preferred)
FentanylOpioid AnalgesicMu-opioid receptor agonist → CNS/PNS pain modulation; minimal histamine releasePain management, RSI adjunct
MorphineOpioid AnalgesicMu-opioid receptor agonist; also causes histamine release → vasodilationPain (controversial in ACS)
NaloxoneOpioid AntagonistCompetitive mu-opioid receptor antagonist → reverses opioid-induced CNS/respiratory depressionOpioid overdose reversal
AlbuterolBeta-2 Agonist / BronchodilatorSelective Beta-2 adrenergic agonist → bronchial smooth muscle relaxation → bronchodilationAsthma, bronchospasm
IpratropiumAnticholinergic BronchodilatorMuscarinic ACh receptor antagonist → reduces bronchoconstriction and secretionsCOPD, asthma (combined with albuterol)
NitroglycerinNitrate / VasodilatorConverts to nitric oxide → activates guanylyl cyclase → smooth muscle relaxation → venodilationACS, hypertensive emergency, pulmonary edema
AspirinAntiplatelet / NSAIDIrreversibly inhibits COX-1 and COX-2 → reduces thromboxane A2 → inhibits platelet aggregationACS
Sodium BicarbonateAlkalinizing AgentProvides HCO3- → buffers H+ → raises blood pH; narrows QRS in Na+ channel blockadeTCA OD, severe hyperkalemia, metabolic acidosis
Calcium ChlorideElectrolyte / Membrane StabilizerRaises extracellular Ca2+ → stabilizes cardiac cell membrane threshold potentialCCB OD, hyperkalemia, hypocalcemia
Dextrose 50%Hypertonic Glucose SolutionProvides exogenous glucose → rapidly raises blood glucose; insulin-independent uptakeHypoglycemia
GlucagonPancreatic HormoneGlucagon receptor agonist → raises cAMP independently of beta receptors → increased HR/contractilityBeta-blocker OD, hypoglycemia
Thiamine (B1)Water-Soluble VitaminCofactor for pyruvate dehydrogenase and alpha-ketoglutarate dehydrogenase → aerobic metabolismWernicke encephalopathy, alcohol withdrawal
OxytocinUterotonic HormoneBinds oxytocin receptors on uterine smooth muscle → myometrial contractionPostpartum hemorrhage (uterine atony)
Mag Sulfate (OB)CNS Depressant / TocolyticNMDA receptor blockade → CNS depression → seizure prophylaxis; also relaxes smooth muscleEclampsia/preeclampsia, preterm labor
Atropine (Organophos)Anticholinergic AntidoteCompetitive muscarinic antagonist → blocks SLUDGE symptoms (secretions, bradycardia, bronchospasm)Organophosphate/nerve agent poisoning