🫁 Airway Emergencies

Master Every
Airway Call.

Universal algorithm, upper & lower airway emergencies, capnography mastery, difficult airway assessment, RSI, and pediatric differences. Built from your paramedic field notes.

9 Sections Covered
LEMON Β· MOANS Β· DOPE Mnemonics
NREMT Exam Tips
BLS before ALS Always
Airway Escalation
1. BVMAlways try first
2. SGA / LMABVM inadequate
3. ETIDefinitive
4. Cric (CICO)Last resort only
EtCO2 Normal35–45 mmHg
ROSC SpikeSudden rise β†’ check pulse
All
πŸ”΄ Upper Airway
🫁 Lower Airway
πŸ’¨ Ventilatory Failure
πŸ“Š Capnography
⚠️ Difficult Airway
πŸ‘Ά Pediatric
πŸ“ NREMT Tips
14 topics
πŸ”„
Universal Airway Algorithm
Apply this sequence to EVERY airway call β€” BLS before ALS
Do This Every Time BLS First
β–Ό
7-step sequence from scene size-up to advanced airway escalation. Never rush past a working BVM.
Step-by-Step Protocol
  • 1
    Scene + Primary AssessmentIdentify "airway NOW" patients: unable to speak, silent chest, cyanosis, stridor at rest, severe retractions, altered mentation, extreme RR with fatigue, poor chest rise
  • 2
    Position + Manual ManeuverHead-tilt/chin-lift (no trauma) OR jaw-thrust (trauma/C-spine concern). Sniffing position aligns oral-pharyngeal-laryngeal axes.
  • 3
    SuctionGurgling = suction FIRST! Limits: ≀15 sec adult, ≀10 sec child, ≀5 sec infant. Suction on the way OUT. Pre-oxygenate when possible.
  • 4
    Basic AdjunctsOPA: unconscious, NO gag reflex. NPA: gag present or OPA not tolerated; avoid with facial/head trauma or suspected basilar skull fracture.
  • 5
    Oxygenate + VentilateTwo-person BVM preferred. Ventilate to visible chest rise. Avoid excessive volume/pressure (especially peds). Use capnography.
  • 6
    CPAP (when appropriate)Breathing but failing oxygenation/ventilation (CHF, COPD, asthma with adequate mentation). Monitor for hypotension/barotrauma.
  • 7
    Escalate AirwayIf BVM/CPAP inadequate β†’ SGA β†’ ETI β†’ Cric (CICO only). Never rush past a working BVM.
Airway Sound Recognition
SoundMeaningImmediate Action
SnoringTongue/soft tissue obstructionReposition, jaw thrust, OPA/NPA
GurglingFluid in airwaySUCTION IMMEDIATELY
Stridor (inspiratory)Upper airway narrowingEmergency β€” act fast, risk of complete obstruction
Wheeze (expiratory)Lower airway/bronchospasmBronchodilators, CPAP
Barking coughCroup β€” viral subglottic edemaMinimize agitation, blow-by O2, neb epi if severe
Silent chestNear-complete obstruction OR severe failureCRITICAL β€” imminent arrest
Hoarse/muffled voiceEpiglottitis, angioedema, burnsDo NOT manipulate β€” prepare for abrupt closure
πŸ”΄
CRITICAL RULE: If you can oxygenate and ventilate with BVM, do NOT rush to an advanced airway. Optimize BVM first.
πŸ”΄
Upper Airway Obstruction
Tongue obstruction, FBAO, Croup β€” stridor is time critical
Upper Airway Stridor = Emergency
β–Ό
Stridor indicates narrowed upper airway. Anticipate sudden complete obstruction. Treat as emergency.
1A β€” Tongue / Soft Tissue Obstruction
Pathophysiology: Loss of muscle tone β†’ tongue falls back β†’ occludes posterior pharynx.
Signs: Snoring respirations, poor air movement, improves with repositioning.
  • 1
    Position + manual maneuver (head-tilt/chin-lift or jaw-thrust)
  • 2
    Suction as needed
  • 3
    OPA if no gag / NPA if gag present (no contraindication)
  • 4
    BVM with high-flow O2 + capnography
  • 5
    If unable to maintain: escalate to advanced airway per protocol
1B β€” Foreign Body Airway Obstruction (FBAO)
Partial: coughing, wheeze/stridor with some air movement  |  Complete: cannot speak/cough, silent efforts, rapid hypoxia, cyanosis
  • 1
    Conscious: age-appropriate obstruction maneuvers (Heimlich/back blows per protocol)
  • 2
    Worsening/unresponsive: open airway, suction, attempt ventilation
  • 3
    Direct visualization with laryngoscope β†’ remove with Magill forceps (especially peds)
  • 4
    If not removable: attempt intubation with smaller tube to push obstruction down
  • 5
    CICO: rescue airway / needle cricothyrotomy per protocol
1C β€” Croup (Viral Subglottic Edema)
Pathophysiology: Parainfluenza β†’ subglottic edema β†’ barking cough + stridor. Peak age 6mo–3yr. Worse at night.
Signs: Barking "seal" cough, inspiratory stridor, retractions, low-grade fever after URI.
  • 1
    Keep child CALM β€” agitation worsens obstruction. Position of comfort.
  • 2
    Blow-by oxygen if tolerated (NRB may increase agitation)
  • 3
    Nebulized racemic epinephrine for moderate/severe stridor at rest per protocol
  • 4
    Corticosteroid per protocol (dexamethasone β€” reduces edema, onset delayed)
  • 5
    Prepare to ventilate if fatigue/deterioration; transport and reassess frequently
⚠️
NREMT ALERT: Croup gets racemic epinephrine for respiratory distress β€” NOT a tongue depressor, NOT oral airway manipulation.
🚨
Epiglottitis & Angioedema
Supraglottic emergencies β€” DO NOT manipulate the airway
Upper Airway No Tongue Depressor
β–Ό
Bacterial or allergic-mediated supraglottic swelling with risk of sudden complete obstruction. Keep calm, don't agitate.
1D β€” Epiglottitis β€” The "4 D's"
Pathophysiology: Bacterial infection (H. influenzae or Strep) β†’ swollen epiglottis β†’ risk of sudden complete obstruction. Now most common in adults.
D
Drooling
D
Dysphagia
Can't swallow
D
Dysphonia
Hot potato voice
D
Distress
Tripod, toxic look
  • 1
    CALM environment β€” do NOT agitate the patient
  • 2
    Do NOT inspect oropharynx β€” no tongue depressor (can trigger spasm/complete obstruction)
  • 3
    Do NOT insert OPA or routine oral devices
  • 4
    Blow-by oxygen ONLY if patient tolerates β€” do not force a mask
  • 5
    BVM + suction + backup airway ready for abrupt closure
  • 6
    Rapid transport; advanced airway only by most experienced provider with surgical cric backup
πŸ”΄
NEVER use a tongue depressor in suspected epiglottitis β€” this can trigger complete obstruction and death.
1E β€” Anaphylaxis / Angioedema
Pathophysiology: IgE-mediated mast cell degranulation β†’ tongue/laryngeal edema + bronchospasm + vasodilation. Progression can be RAPID β€” minutes to complete obstruction.
Signs: Facial/tongue swelling, hoarseness, stridor, wheeze, urticaria, hypotension, tachycardia.
  • 1
    Epinephrine IM immediately β€” 1:1,000, 0.3–0.5mg IM anterior/lateral thigh. This is the primary lifesaving drug.
  • 2
    High-flow O2; continuous monitoring (SpO2, ECG, capnography); prepare for rapid deterioration
  • 3
    Repeat epinephrine in 5–15 min if no improvement per protocol
  • 4
    Add-ons: diphenhydramine, corticosteroids, albuterol for bronchospasm, IV fluids/vasopressors for shock
  • 5
    Intubation with EXTREME caution β€” edema distorts anatomy; use smaller-than-expected tube; video laryngoscopy if available
  • 6
    If CICO: SGA attempt, then cricothyrotomy per protocol
πŸ”₯
Airway Burns / Inhalation Injury
Progressive edema β€” secure airway EARLY before swelling closes it
Upper Airway Time Critical
β–Ό
Thermal/chemical irritation causes progressive airway edema that worsens over hours. CO and cyanide poisoning possible.
Signs & Symptoms
  • Facial burns, singed nasal/facial hair, soot in nares/oropharynx
  • Carbonaceous (black) sputum β€” hallmark of inhalation injury
  • Hoarseness, stridor, progressive swelling
  • Cherry-red skin (CO) or altered mentation (CO/cyanide)
Treatment β€” First to Last
  • 1
    High-flow 100% O2 via NRB immediately β€” reduces CO half-life
  • 2
    TIME-CRITICAL β€” edema will worsen; early airway control before swelling closes it
  • 3
    Suction as needed; plan early advanced airway with smaller tube if edema present
  • 4
    Cyanide antidote per protocol if suspected (hydroxocobalamin)
  • 5
    Progressing obstruction beyond intubation ability β†’ cricothyrotomy per protocol
🫁
Asthma & COPD Exacerbation
Lower airway obstruction β€” watch for air trapping, rising EtCO2, silent chest
Lower Airway Shark Fin Waveform
β–Ό
Bronchospasm + air trapping β†’ increased WOB. DANGER: silent chest, rising EtCO2, fatigue = pre-arrest.
2A β€” Asthma / Status Asthmaticus
Pathophysiology: Bronchial smooth muscle spasm + mucosal inflammation + mucus plugging β†’ air trapping β†’ increased WOB. Status asthmaticus = refractory to standard treatment.
Capnography: "Shark fin" waveform (slow obstructed exhalation). Rising EtCO2 in tiring patient = pre-arrest.
  • 1
    O2 + continuous monitoring (SpO2, capnography, cardiac)
  • 2
    Nebulized albuterol per protocol β€” repeat as needed
  • 3
    Add ipratropium (moderate-severe) β€” synergistic with albuterol
  • 4
    Corticosteroid early (methylprednisolone/dexamethasone) β€” onset delayed
  • 5
    CPAP if persistent bronchospasm and patient can protect airway
  • 6
    Magnesium sulfate IV for severe/refractory bronchospasm per protocol
  • 7
    If tiring/failing: SLOW rate BVM, allow FULL exhalation, prepare advanced airway
⚠️
ASTHMA VENTILATION: Avoid rapid/aggressive bagging β€” worsens air trapping (auto-PEEP) and causes hypotension. Use slow rate (6–8 bpm), long expiratory time (I:E = 1:3 or longer).
2B β€” COPD Exacerbation
Pathophysiology: Chronic airflow limitation + acute trigger β†’ ventilation failure and CO2 retention.
Signs of CO2 retention: Headache, flushing, altered mentation, asterixis (flapping tremor).
  • 1
    Controlled O2 titrated to SpO2 88–92% in known COPD β€” but NEVER withhold O2 if hypoxic
  • 2
    Bronchodilators (albuterol + ipratropium) + corticosteroids per protocol
  • 3
    CPAP β€” watch for hypotension and barotrauma
  • 4
    If failing: BVM ventilatory support and advanced airway per protocol
πŸ’¨
Pulmonary Edema & Opioid OD
CHF respiratory failure and ventilatory depression β€” know the priorities
Lower Airway Ventilatory Failure
β–Ό
Pulmonary edema: CPAP is the primary intervention. Opioid OD: BVM FIRST, then naloxone β€” not the other way around.
2C β€” Pulmonary Edema / CHF
Pathophysiology: Fluid in alveoli β†’ diffusion failure β†’ severe hypoxia.
Signs: Severe dyspnea, orthopnea, bilateral crackles, pink frothy sputum, JVD, peripheral edema, hypoxia.
  • 1
    Sit patient upright β€” legs dependent reduces preload
  • 2
    High-flow O2 + continuous monitoring
  • 3
    CPAP early if tolerated β€” PEEP recruits alveoli, pushes fluid back, reduces WOB
  • 4
    Nitroglycerin per protocol if hypertensive (preload reduction)
  • 5
    Diuretics per protocol if indicated; BVM/advanced airway if failing
3A β€” Opioid Overdose / Respiratory Depression
Signs: Slow/shallow respirations or apnea, altered MS, pinpoint pupils (KEY), possible hypotension, track marks.
Key point: EtCO2 rises BEFORE SpO2 drops β€” capnography catches respiratory depression early.
  • 1
    Open airway + BVM with O2 FIRST β€” do NOT wait for naloxone
  • 2
    Naloxone per protocol β€” titrate to adequate respirations, NOT full wakefulness (avoid acute withdrawal)
  • 3
    Suction ready β€” anticipate vomiting on waking; consider recovery position
  • 4
    Monitor for re-sedation (naloxone wears off faster than most opioids) β€” repeat dosing as needed
πŸ”΄
BVM BEFORE NALOXONE β€” fix oxygenation first. Naloxone without ventilation doesn't save a hypoxic brain.
πŸ“Š
Capnography Master Reference
EtCO2 normal 35–45 mmHg β€” measures ventilation, perfusion, and metabolism
Capnography Gold Standard Confirmation
β–Ό
Waveform capnography is the most important airway monitoring tool available. Catches problems before SpO2 changes.
EtCO2 Values & Meanings
ValueMeaning
35–45 mmHgNormal β€” adequate ventilation
<35 mmHgHyperventilation, decreased perfusion (shock, PE, cardiac arrest)
>45 mmHgHypoventilation, COPD/asthma air trapping, rising ICP
Absent waveformEsophageal intubation, cardiac arrest with no CPR, circuit disconnection
<10 mmHg during CPRInadequate compression quality β€” switch compressors
Sudden spike to 35–45ROSC β€” check pulse BEFORE stopping CPR
Waveform Pattern Interpretation
PatternClinical Meaning
Normal rectangleAdequate ventilation and airway patency confirmed
"Shark fin" (sloped plateau)Lower airway obstruction: asthma, COPD, bronchospasm
Elevated baselineIncomplete exhalation / rebreathing CO2 β€” extend expiratory time
Sudden waveform lossCheck pulse FIRST (cardiac arrest?), THEN confirm tube position
Gradually rising trendCO2 retention: hypoventilation, tiring patient, increasing air trapping
Gradually falling trendImproving ventilation OR decreasing cardiac output β€” reassess
Bumps during plateauPatient breathing against ventilation or inadequate sedation
ETT Confirmation Protocol
  • Gold standard: Continuous waveform capnography showing consistent rectangular waveform after intubation
  • Colorimetric detector: ALWAYS confirm with at least 6 ventilations; can give false negative in low-flow states
  • Confirm after EVERY patient move: tube position + chest rise + breath sounds + waveform
  • Sudden waveform loss post-intubation: check pulse first, then confirm tube position (DOPE)
⚠️
Difficult Airway: LEMON & MOANS
Predict difficult intubation and difficult BVM before every attempt
Difficult Airway NREMT Favorite
β–Ό
Use LEMON before every advanced airway attempt to anticipate problems and prepare backup plans.
LEMON β€” Predict Difficult Intubation
L
Look Externally
Facial trauma, obesity, short neck, beard, large tongue, goiter
E
Evaluate 3-3-2
3 fingers mouth | 3 chin to hyoid | 2 hyoid to thyroid
M
Mallampati
Class I (all visible) β†’ Class IV (hard palate only)
O
Obstruction
Angioedema, epiglottitis, FB, burns, tumor
N
Neck Mobility
C-collar, arthritis, obesity limit extension
S
Saturation
SpO2 100% = reserve | <90% = no reserve, act fast
MOANS β€” Predict Difficult BVM
M
Mask Seal
Beard, facial trauma, obesity, absent teeth
O
Obstruction
Oropharyngeal obstruction, morbid obesity
A
Age
Elderly (decreased compliance); infants (different anatomy)
N
No Teeth
Paradoxically makes BVM seal harder β€” leave dentures in if stable
S
Stiff / Snoring
Stiff lungs (fibrosis, ARDS), snoring (tongue obstruction)
Advanced Airway Escalation Sequence
  • 1
    Optimize BVM: two-person seal, OPA/NPA, suction, PEEP valve β€” preoxygenate 3+ min
  • 2
    SGA/LMA/i-gel/King LT if BVM inadequate or intubation delayed/failed
  • 3
    ETI β€” BURP if needed, Cormack-Lehane grading, confirm with waveform capnography
  • 4
    CICO β†’ Cricothyrotomy β€” last resort if all above fail
πŸ’‰
RSI β€” Rapid Sequence Intubation
Induction agent + NMBA for optimal intubating conditions
Advanced Airway Succinylcholine Contraindications
β–Ό
RSI = concurrent induction agent + neuromuscular blocking agent. Used when airway cannot wait and patient is not apneic/unconscious.
RSI Phase Sequence
PhaseKey Actions
PreparationLEMON assessment, equipment ready, backup plan, CICO kit available
Preoxygenation3–5 min high-flow O2 (NRB + NC at 15 LPM); creates O2 reserve β€” extends safe apneic time
PretreatmentLidocaine (ICP concern), atropine (peds bradycardia prevention) β€” per protocol
Induction AgentSedative/hypnotic (ketamine, etomidate) β€” provides unconsciousness per protocol
Paralytic (NMBA)Succinylcholine (fast onset/offset) or Rocuronium (non-depolarizing) β€” per protocol
IntubationLaryngoscopy; BURP if needed; Grade 1–4 (Grade 1 = full glottis view)
ConfirmationWaveform capnography + chest rise + breath sounds
Post-IntubationSedation/analgesia; SpO2, EtCO2, vitals; secure tube; reassess after every move
Cricothyrotomy (CICO β€” Last Resort)
  • Indication: CANNOT INTUBATE + CANNOT OXYGENATE/VENTILATE by any other method
  • Anatomy: Cricothyroid membrane β€” inferior to thyroid cartilage, superior to cricoid ring ("soft spot")
  • Warning: Complete obstruction above insertion site can prevent exhalation β€” risk of hypercarbia/barotrauma
  • Peds: Reserved for complete occlusion when NO other method works β€” needle technique preferred in young children
⚠️
SUCCINYLCHOLINE CONTRAINDICATIONS: Hyperkalemia risk with burns >48h, crush injury, prolonged immobility, denervation injuries, renal failure. Know your local protocol.
πŸ‘Ά
Pediatric Airway
Children are NOT small adults β€” key anatomic and physiologic differences
Pediatric Bradycardia = Hypoxia
β–Ό
Higher metabolic rate, less reserve, and different anatomy mean kids decompensate rapidly and without warning.
Anatomic Differences vs Adult
FeaturePediatric Difference & Implication
Large OcciputNeutral/sniffing position achieved more easily; avoid over-extension in infants
Large TongueLarger relative to oral cavity β†’ more easily obstructs airway
EpiglottisSofter, omega-shaped, more horizontal β†’ Miller blade often preferred
Narrowest PointCricoid ring (subglottic) in <8 yrs vs glottic in adults β†’ uncuffed tubes in young children
Small AirwayMinor edema = MAJOR resistance increase (resistance ∝ 1/radius⁴)
Respiratory ReserveHigher metabolic rate β†’ desaturate MUCH faster than adults
BradycardiaLate/OMINOUS sign of hypoxia β€” do NOT wait; act immediately
ETT Size Quick Reference
Age GroupETT Size (uncuffed)Formula
Newborn/Neonate3.0–3.5 mm(AgeΓ·4) + 4
Infant (1–12 mo)3.5–4.0 mm
Toddler (1–3 yr)4.0–4.5 mm
Preschool (3–5 yr)4.5–5.0 mm
School age (6–12 yr)5.0–6.5 mm
Adolescent (>12 yr)Adult sizing
Key Peds Pearls
  • Treat respiratory distress EARLY β€” kids decompensate rapidly and without warning
  • AVOID agitation in croup and epiglottitis β€” crying/agitation worsens obstruction
  • Suction causes vagal bradycardia β€” limit duration, re-oxygenate immediately
  • Gastric distention most common secondary cause when ventilations suddenly become difficult β†’ insert NG/OG tube
  • Use Broselow tape or length-based estimation for drug and equipment sizing
πŸ”΄
DOPE β€” Sudden Deterioration in Intubated Patient
Work through this systematically any time an intubated patient worsens
Intubated Patient NREMT High-Yield
β–Ό
D β€” Displacement | O β€” Obstruction | P β€” Pneumothorax | E β€” Equipment failure. Check in order.
DOPE Assessment
ProblemSignsFix
D β€” DisplacementAbsent/unequal breath sounds, no waveform, epigastric soundsVisualize tube, confirm with capnography, re-intubate if needed
O β€” ObstructionIncreased resistance, poor chest rise, secretions, high peak pressuresSuction tube, check for kinking, consider bite block
P β€” PneumothoraxAbsent unilateral breath sounds, hypotension, JVD, tracheal deviationNeedle decompression (2nd ICS MCL or 4th/5th ICS AAL)
E β€” Equipment FailureBVM malfunction, O2 supply failure, tube leak, circuit disconnectionSwitch equipment, check O2 supply, verify connections
πŸ“‹
All Airway Mnemonics
LEMON Β· MOANS Β· RODS Β· SMART Β· DOPE Β· BURP Β· SALAD Β· CICO
NREMT Favorites Quick Reference
β–Ό
Complete mnemonic master list for NREMT exam and field reference.
MnemonicMeaning & Use
LEMONDifficult Intubation: Look | Evaluate 3-3-2 | Mallampati | Obstruction | Neck mobility (+S for Saturation)
MOANSDifficult BVM: Mask seal | Obstruction/Obesity | Age | No teeth | Stiff/Snoring lungs
RODSDifficult SGA: Restricted mouth | Obstruction | Distorted airway | Stiff lungs
SMARTDifficult Cric: Surgery/scarring | Mass/Tumor | Access | Radiation | Thick neck
DOPESudden deterioration intubated patient: Displacement | Obstruction | Pneumothorax | Equipment failure
BURPImprove laryngoscopic view: Backward | Upward | Right | Pressure on thyroid cartilage
4 DsEpiglottitis: Drooling | Dysphagia | Dysphonia | Distress
SALADContaminated airway: Suction Assisted Laryngoscopy and Airway Decontamination
CICOCannot Intubate Cannot Oxygenate β†’ triggers surgical airway
πŸ“
NREMT Exam Strategy & High-Yield Facts
Common exam traps and the correct thinking for airway questions
NREMT 2024+ Exam Traps
β–Ό
Airway makes up 8–12% of NREMT questions. Clinical judgment is the largest domain β€” most questions test decision-making.
Exam Traps & Correct Thinking
  • Epiglottitis: NEVER tongue depressor, NEVER OPA. Answer = keep calm, blow-by O2, rapid transport.
  • Croup with severe stridor at rest: Answer = racemic epinephrine. NOT cool mist on NREMT.
  • Asthma silent chest: Near-complete obstruction = CRITICAL. Do NOT celebrate β€” escalate immediately.
  • COPD O2: Target SpO2 88–92% β€” but NEVER withhold O2 from a hypoxic patient.
  • Anaphylaxis: Epinephrine is ALWAYS first and primary. Not diphenhydramine.
  • Opioid OD: BVM FIRST, THEN naloxone. Not naloxone first.
  • Gurgling sound: SUCTION first β€” not OPA, not BVM.
  • OPA contraindicated: Gag reflex present β†’ use NPA instead.
  • NPA contraindicated: Suspected basilar skull fracture or significant facial/head trauma.
  • ETT confirmation: Continuous waveform capnography is the GOLD STANDARD.
  • Rising EtCO2 in intubated patient: Hypoventilation or fatigue β€” increase rate/depth.
  • EtCO2 suddenly zero post-intubation: Check pulse FIRST (cardiac arrest?), THEN confirm tube.
High-Yield Numbers
ValueWhat It Represents
35–45 mmHgNormal EtCO2 range
<10 mmHg during CPRPoor CPR quality β€” improve compressions or switch
Sudden spike to 35–45ROSC β€” check pulse before stopping CPR
SpO2 88–92%COPD target oxygenation
SpO2 94–99%Standard target for most adult patients
≀15 secMaximum suction time for adults
≀10 secMaximum suction time for children
≀5 secMaximum suction time for infants
10–12 bpmAdult ventilation rate with advanced airway in place
0.3–0.5 mg IMAdult epinephrine dose for anaphylaxis (1:1,000, anterolateral thigh)
βœ…
Universal Rules β€” Never Forget
Always Β· Never Β· Watch For β€” rapid review before any airway call
Rapid Review Core Rules
β–Ό
The foundational rules that govern every airway decision. BLS before ALS β€” always.
βœ…
ALWAYS: BLS before ALS. Suction before adjuncts. Preoxygenate before any advanced attempt. Confirm ETT with waveform capnography. DOPE for every sudden deterioration.
πŸ”΄
NEVER: Tongue depressor in epiglottitis. Fast bagging in asthma. Naloxone before BVM in opioid OD. Force mask on epiglottitis/croup. Amiodarone for Torsades.
⚠️
WATCH FOR: Silent chest (asthma near-arrest). Sudden EtCO2 loss (tube displacement or arrest). Peds bradycardia (hypoxia, not primary cardiac). Rising EtCO2 in intubated patient (hypoventilation). RV infarct with inferior STEMI (hold nitro).
Condition Snapshot
ConditionKey SignFirst Action
Tongue obstructionSnoringReposition + jaw thrust
FBAO consciousSilent, can't speakHeimlich/back blows
CroupBarking cough + stridorCalm + blow-by O2
EpiglottitisDrooling + tripod + hoarseCalm β€” NO manipulation
AnaphylaxisFacial swelling + wheezeEpi 0.3–0.5mg IM NOW
Airway burnSoot + hoarseness100% O2 + early ETI
Asthma severeWheeze β†’ silent chestAlbuterol + CPAP
COPD exac.Wheeze + CO2 signsControlled O2 88–92%
Pulm. edemaCrackles + frothy sputumUpright + CPAP
Opioid ODMiosis + slow RRBVM FIRST then Narcan